CRISPR-Cas systems, Drosophila, growth, insulin, metabolomics, multi-omics, omics, phosphoproteomics, protein-protein interactions, Systems Biology
Drosophila, p53, AMPK, Upd2/Leptin, insulin-producing cells, fat body, metabolism
Drosophila, tumor-induced wasting, cancer cachexia, Upd3, ImpL2, Jak/Stat signaling, muscle, fat body, metabolism, lipid loss, hyperglycemia