Author Page for Hulun Li

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GM-CSF-Producing B Cells Regulate Microglial Activation and Promote Microglial Transition to an Inflammation State in Experimental Autoimmune Encephalomyelitis Model Mice

Number of pages: 44 Posted: 11 Jun 2019

Xi Wang, Zhongze He, Siying Qu, Wantong Zhai, Jiahui Zhu, Xiaoyi Zou, Xiujuan Lang, Shenyue Wang, Zhaoying Li, Guangyou Wang, Jiangtian Tian, Shaohong Fang, Bo Sun and Hulun Li

affiliation not provided to SSRN, affiliation not provided to SSRN, affiliation not provided to SSRN, affiliation not provided to SSRN, affiliation not provided to SSRN, affiliation not provided to SSRN, affiliation not provided to SSRN, affiliation not provided to SSRN, affiliation not provided to SSRN, affiliation not provided to SSRN, affiliation not provided to SSRN, affiliation not provided to SSRN, affiliation not provided to SSRN and Harbin Medical University

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Citation 1

Abstract:

Methotrexate Therapy to Promote Endothelialization and Stability of Neointima by Reducing Inflammation Activation in a Rabbit Stenting Model

Number of pages: 42 Posted: 10 Nov 2018

Ruoxi Zhang, Xianglan Liu, Yong Sun, Jian Wu, Maomao Zhang, Jinwei Tian, Shaohong Fang, Yang Zheng, Hulun Li, Jingbo Hou and Bo Yu

Harbin Medical University, Harbin Medical University, Second Affiliated Hospital, Department of Cardiology, Harbin Medical University, Harbin Medical University, Harbin Medical University, Harbin Medical University, Harbin Medical University, Harbin Medical University, Harbin Medical University, Harbin Medical University, Second Affiliated Hospital, Department of Cardiology and Harbin Medical University, Second Affiliated Hospital, Department of Cardiology

Downloads 54 (1,020,567)

Hulun Li

Harbin Medical University

2

130

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Scholarly Papers (2)

GM-CSF-Producing B Cells Regulate Microglial Activation and Promote Microglial Transition to an Inflammation State in Experimental Autoimmune Encephalomyelitis Model Mice

Abstract:

Methotrexate Therapy to Promote Endothelialization and Stability of Neointima by Reducing Inflammation Activation in a Rabbit Stenting Model

Abstract: