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Francis Palisson

Fundación DEBRA Chile

Santiago

Chile

SCHOLARLY PAPERS

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Scholarly Papers (1)

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Epithelial HMGB1 Delays Skin Wound Healing and Drives Tumor Initiation by Priming Neutrophils to NETosis

Number of pages: 35 Posted: 05 Feb 2019
Esther Hoste, Lisette van Hove, Leen Catrysse, Hanna-­Kaisa Vikkula, Ria Roelandt, Wim Declercq, Ignacia Fuentes, Francis Palisson, Sergio Gonzalez, Julio C. Salas-­Alanis, Peter Huebener, Robert F. Schwabe and Geert van Loo
VIB Center for Inflammation Research - Unit for Cellular and Molecular Pathophysiology, VIB Center for Inflammation Research - Unit for Cellular and Molecular Pathophysiology, VIB Center for Inflammation Research - Unit for Cellular and Molecular Pathophysiology, VIB Center for Inflammation Research - Unit for Cellular and Molecular Pathophysiology, VIB Center for Inflammation Research - Molecular Signaling and Cell Death Unit, VIB Center for Inflammation Research - Molecular Signaling and Cell Death Unit, Fundación DEBRA Chile, Fundación DEBRA Chile, Pontificia Universidad Católica de Chile - Department of Pathology, Dystrophic Epidermolysis Bullosa Research Association Mexico, University of Hamburg - Department of Internal Medicine, Columbia University - Department of Medicine and VIB Center for Inflammation Research - Unit for Cellular and Molecular Pathophysiology
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tumor microenvironment, skin inflammation, innate immunity, NETosis, HMGB1, wound healing