Joanna Zyla

Max Planck Institute for Infection Biology - Department of Immunology

Germany

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Scholarly Papers (1)

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The Endogenous Metabolite Sphingosine-1-Phosphate (S1P) Activates NOD1/2-Dependent Inflammation Triggered by Cellular Stress

Number of pages: 71 Posted: 19 May 2020
Max Planck Institute for Infection Biology - Department of Immunology, Max Planck Institute for Infection Biology - Department of Immunology, Chinese Academy of Sciences - State Key Laboratory of Magnetic Resonance and Atomic Molecular Physics, Max Planck Institute for Infection Biology - Department of Immunology, Max Planck Institute for Infection Biology - Department of Immunology, Max Planck Institute for Infection Biology - Department of Immunology, Max Planck Institute for Infection Biology - Department of Immunology, Max Planck Institute for Infection Biology - Department of Immunology, Max Planck Institute for Infection Biology - Microarray Core Facility, University of Hohenheim - Institute of Nutritional Medicine, University of Hohenheim - Institute of Nutritional Medicine, Institute of Immunology, Institute of Immunology, Institut Pasteur - Department of Microbiology, Biology and Genetics of the Bacterial Cell Wall, University of Hohenheim - Institute of Nutritional Medicine, Max Planck Institute for Infection Biology - Department of Immunology and Max Planck Institute for Infection Biology - Department of Immunology
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Abstract:

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Nod-like receptors, NOD1/2, Innate immunity, Inflammation, Sphingolipid metabolism, Sphingosine-1-phosphate, Cellular stress