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Yunzeng Zou

Fudan University - Shanghai Institute of Cardiovascular Diseases

China

SCHOLARLY PAPERS

4

DOWNLOADS

332

TOTAL CITATIONS

1

Scholarly Papers (4)

TAK1 Activation by NLRP3 Deficiency Confers Cardioprotection Against Pressure Overload-Induced Cardiomyocyte Pyroptosis and Hypertrophy

Number of pages: 59 Posted: 04 Nov 2022
Fudan University - Shanghai Institute of Cardiovascular Diseases, Shanghai Jiao Tong University (SJTU) - Department of Cardiovascular Medicine, Fudan University - Shanghai Institute of Cardiovascular Diseases, Fudan University - Shanghai Institute of Cardiovascular Diseases, Guangdong Laboratory Animals Monitoring Institute, Shanghai Jiao Tong University (SJTU) - Department of Cardiovascular Medicine, Fudan University - Shanghai Institute of Cardiovascular Diseases, Fudan University - Shanghai Institute of Cardiovascular Diseases, Fudan University - Shanghai Institute of Cardiovascular Diseases, Fudan University - Cardiovascular Department, Guangzhou Medical University - China State Key Laboratory of Respiratory Disease & National Clinical Research Center for Respiratory Disease, Fudan University - Shanghai Institute of Cardiovascular Diseases, Tongji University - Shanghai East Hospital, York University - Department of Biology, Fudan University - Shanghai Institute of Cardiovascular Diseases, Fudan University - Shanghai Institute of Cardiovascular Diseases, Fudan University - Shanghai Institute of Cardiovascular Diseases and Fudan University - Shanghai Institute of Cardiovascular Diseases
Downloads 85 (779,641)

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cardiac remodeling, cell death, hypertrophy, pressure overload, pyroptosis

TAK1 Activation by NLRP3 Deficiency Confers Cardioprotection Against Pressure Overload-Induced Cardiomyocyte Pyroptosis and Hypertrophy

Number of pages: 83 Posted: 13 Feb 2023
Fudan University - Shanghai Institute of Cardiovascular Diseases, Shanghai Jiao Tong University (SJTU) - Department of Cardiovascular Medicine, Fudan University - Shanghai Institute of Cardiovascular Diseases, Fudan University - Shanghai Institute of Cardiovascular Diseases, Guangdong Laboratory Animals Monitoring Institute, Shanghai Jiao Tong University (SJTU) - Department of Cardiovascular Medicine, Fudan University - Shanghai Institute of Cardiovascular Diseases, Fudan University - Shanghai Institute of Cardiovascular Diseases, Fudan University - Shanghai Institute of Cardiovascular Diseases, Fudan University - Cardiovascular Department, Guangzhou Medical University - China State Key Laboratory of Respiratory Disease & National Clinical Research Center for Respiratory Disease, Fudan University - Shanghai Institute of Cardiovascular Diseases, Tongji University - Shanghai East Hospital, York University - Department of Biology, Fudan University - Shanghai Institute of Cardiovascular Diseases, Fudan University - Shanghai Institute of Cardiovascular Diseases, Fudan University - Shanghai Institute of Cardiovascular Diseases and Fudan University - Shanghai Institute of Cardiovascular Diseases
Downloads 76 (848,302)

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cardiac remodeling, cell death, hypertrophy, pressure overload, pyroptosis

2.

Elevated Wnt2 and Wnt4 Activate NF-κB Signaling to Promote Cardiac Fibrosis by Cooperation of Fzd4/2 and LRP6 Following Myocardial Infarction

Number of pages: 48 Posted: 07 Jun 2021
Fudan University - Shanghai Institute of Cardiovascular Diseases, Capital Medical University - Beijing Friendship Hospital, Fudan University - Shanghai Institute of Cardiovascular Diseases, Fudan University - Shanghai Institute of Cardiovascular Diseases, Fudan University - Shanghai Institute of Cardiovascular Diseases, Dalian University - Department of Cardiology, Dalian University - Department of Cardiology, Fudan University - Shanghai Institute of Cardiovascular Diseases, Fudan University - Shanghai Institute of Cardiovascular Diseases, Fudan University - Shanghai Institute of Cardiovascular Diseases, Fudan University - Shanghai Institute of Cardiovascular Diseases, Fudan University - Shanghai Institute of Cardiovascular Diseases, Shanghai Jiao Tong University (SJTU) - School of Medicine, Fudan University - Shanghai Institute of Cardiovascular Diseases, Capital Medical University - Beijing Friendship Hospital and Fudan University - Shanghai Institute of Cardiovascular Diseases
Downloads 102 (679,608)
Citation 1

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Wnt, cardiac fibrosis, Myocardial infarction, Frizzled, NF-κB

3.

Disruption of Histamine-H1r Signaling Exacerbates Cardiac Microthrombosis after Periodontal Disease Via Tlr4/Nfκb-P65 Pathway

Number of pages: 45 Posted: 27 Jun 2023
Fudan University, Fudan University - Shanghai Stomatological Hospital, Fudan University - Shanghai Institute of Cardiovascular Diseases, Shanghai University of Medicine and Health Sciences, Fudan University, Fudan University - Shanghai Institute of Cardiovascular Diseases, Shanghai Jiao Tong University (SJTU), Fudan University - Shanghai Institute of Cardiovascular Diseases, Fudan University - Shanghai Institute of Cardiovascular Diseases, Fudan University - Shanghai Institute of Cardiovascular Diseases, Guangzhou Medical University and Fudan University - Shanghai Institute of Cardiovascular Diseases
Downloads 46 (1,133,231)

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Periodontal inflammation, Cardiac microthrombosis, Histidine decarboxylase, Histamine receptors, neutrophil, NFκB

4.

ORP5 Preserves Mitochondrial Function and Suppresses Apoptosis through Inhibition of VDAC1 Oligomerization in Myocardial Infarction

Number of pages: 35 Posted: 29 Apr 2026
affiliation not provided to SSRN, affiliation not provided to SSRN, affiliation not provided to SSRN, Fudan University - Shanghai Institute of Cardiovascular Diseases, affiliation not provided to SSRN, Fudan University - Shanghai Institute of Cardiovascular Diseases, affiliation not provided to SSRN, affiliation not provided to SSRN, affiliation not provided to SSRN and Fudan University - Shanghai Institute of Cardiovascular Diseases
Downloads 23 (1,436,435)

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Apoptosis, Myocardial infarction, Mitochondrial function, ORP5, Vdac1 oligomerization, Mitochondria-associated endoplasmic reticulum membranes