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Svena Verma

Memorial Sloan Kettering Cancer Center - Ludwig Collaborative and Swim Across America Laboratory

SCHOLARLY PAPERS

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Scholarly Papers (1)

1.

MCT4-Dependent Lactate Secretion Suppresses Antitumor Immunity in LKB1-Deficient NSCLC

Number of pages: 50 Posted: 23 Sep 2022
University of Texas at Houston - Department of Thoracic/Head and Neck Medical Oncology, University of Texas at Houston - Department of Thoracic/Head and Neck Medical Oncology, University of Texas at Houston - Department of Thoracic/Head and Neck Medical Oncology, University of Texas at Houston - Department of Genomic Medicine, University of Texas at Houston - Department of Thoracic/Head and Neck Medical Oncology, University of Texas at Houston - Department of Thoracic/Head and Neck Medical Oncology, University of Texas at Houston - Department of Bioinformatics and Computational Biology, University of Texas at Houston - Department of Bioinformatics and Computational Biology, University of Texas at Houston - Department of Translational Molecular Pathology, University of Texas at Houston - Department of Thoracic/Head and Neck Medical Oncology, University of Texas at Houston - Department of Translational Molecular Pathology, Memorial Sloan Kettering Cancer Center - Ludwig Collaborative and Swim Across America Laboratory, Memorial Sloan Kettering Cancer Center - Ludwig Collaborative and Swim Across America Laboratory, Memorial Sloan Kettering Cancer Center - Ludwig Collaborative and Swim Across America Laboratory, New York University (NYU) - Thoracic Oncology Program, University of Texas at Dallas - Children’s Medical Center Research Institute, University of Texas at Dallas - Hamon Center for Therapeutic Oncology Research, University of Texas at Houston - Department of Genomic Medicine, University of Texas at Houston - Department of Thoracic/Head and Neck Medical Oncology and University of Texas at Houston - Department of Thoracic/Head and Neck Medical Oncology
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Abstract:

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LKB1, lung adenocarcinoma, PD-1, immunotherapy resistance, lactate, MCT4, T cell activation, macrophage polarization, metabolism