William Bateson, Black Slavery, Eugenics and Speciation: The Relative Roles of Politics and Science

Forsdyke, D. R. (2014) PeerJ PrePrints 2:e294v1, The relative roles of politics and science: William Bateson, black slavery, eugenics and speciation

26 Pages Posted: 25 May 2016

Date Written: May 23, 2016

Abstract

The peace of the world, and hence the health of its peoples, are challenged by societal confrontations that can often be labelled “racial” or “ethnic.” Emblematic of this is discrimination based on skin colour. William Bateson’s background and training suggest sympathy with the black emancipation movement. Yet the movement’s success is attributed more to battles between political figures than between scientists with contending views on the biology of racial differences. However, in the long term, Bateson’s contributions to slavery and eugenic issues may be seen as no less important than those of politicians. Mendel’s discovery of what we now know as “genes” languished until seized upon by Bateson in 1900. For six exhausting years he struggled to win scientific acceptance of these biological character-determining units. Later, he pressed the Mendelian message home to the general public, opposing simplistic applications of Mendelian principles to human affairs, and arguing that minor genic differences that distinguished races – e.g. skin colour – can seldom initiate new species. Indeed, the spark that initiates a divergence into two species can be non-genic. We are one reproductively isolated population, the human species.

Keywords: black emancipation, civil war, Darwin, genes, hybrid sterility, Mendel, Morton, reproductive isolation, Romanes, speciation

Suggested Citation

Forsdyke, Donald, William Bateson, Black Slavery, Eugenics and Speciation: The Relative Roles of Politics and Science (May 23, 2016). Forsdyke, D. R. (2014) PeerJ PrePrints 2:e294v1, The relative roles of politics and science: William Bateson, black slavery, eugenics and speciation. Available at SSRN: https://ssrn.com/abstract=2783480 or http://dx.doi.org/10.2139/ssrn.2783480

Donald Forsdyke (Contact Author)

Queen's University ( email )

Kingston, Ontario K7L 3N6
Canada

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