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Clinton Bradfield

Government of the United States of America - Laboratory of Immune System Biology

SCHOLARLY PAPERS

2

DOWNLOADS

151

TOTAL CITATIONS

0

Scholarly Papers (2)

1.

Selective Small Molecule Activation of Mannose Receptor CD206 Recalibrates Macrophage Function to Improve Outcomes of Cancer and Inflammatory Diseases

Number of pages: 169 Posted: 09 Sep 2025
Government of the United States of America - National Institutes of Health (NIH), Government of the United States of America - National Institutes of Health (NIH), Government of the United States of America - National Institutes of Health (NIH), Government of the United States of America - National Institutes of Health (NIH), Government of the United States of America - Thoracic Surgery Branch, Government of the United States of America - National Institutes of Health (NIH), Government of the United States of America - National Institutes of Health (NIH), Government of the United States of America - National Institutes of Health (NIH), Government of the United States of America - National Institutes of Health (NIH), Government of the United States of America - National Institutes of Health (NIH), Government of the United States of America - National Institutes of Health (NIH), Government of the United States of America - National Institutes of Health (NIH), Government of the United States of America - National Institutes of Health (NIH), Nagoya City University - Graduate School of Medical Sciences, Government of the United States of America - National Institutes of Health (NIH), Government of the United States of America - National Institutes of Health (NIH), Government of the United States of America - National Institutes of Health (NIH), Government of the United States of America - Frederick National Laboratory for Cancer Research, Government of the United States of America - National Institutes of Health (NIH), Government of the United States of America - National Institutes of Health (NIH), Government of the United States of America - National Institutes of Health (NIH), Government of the United States of America - National Institutes of Health (NIH), Government of the United States of America - National Institutes of Health (NIH), Government of the United States of America - National Institutes of Health (NIH), Government of the United States of America - National Institutes of Health (NIH), Government of the United States of America - National Institutes of Health (NIH), Government of the United States of America - National Institutes of Health (NIH), Government of the United States of America - National Institutes of Health (NIH), Government of the United States of America - Laboratory of Immune System Biology, Government of the United States of America - Frederick National Laboratory for Cancer Research, Government of the United States of America - Frederick National Laboratory for Cancer Research, Government of the United States of America - National Institutes of Health (NIH), Government of the United States of America - National Institutes of Health (NIH), Government of the United States of America - Laboratory of Immune System Biology, Government of the United States of America - National Institutes of Health (NIH), Government of the United States of America - National Institutes of Health (NIH), Government of the United States of America - National Institutes of Health (NIH), Government of the United States of America - National Institutes of Health (NIH), Government of the United States of America - National Institutes of Health (NIH), Nagoya City University - Graduate School of Medical Sciences, Government of the United States of America - National Institutes of Health (NIH), Government of the United States of America - National Institutes of Health (NIH), Government of the United States of America - National Cancer Institute and Independent
Downloads 117 (621,896)

Abstract:

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mannose receptor 1 (MRC1, CD206), CD206 small molecule agonist, tumor associated macrophages, innate immune indolence, macrophage epigenome, CD206 glycosylation, retinal fibrosis, non-alcoholic steatohepatitis (NASH)

2.

Macrophages Regulate Responses to TLR4 Stimulation by Modulating Susceptibility Towards Anti-Inflammatory IL-10

Number of pages: 56 Posted: 03 Mar 2025
Government of the United States of America - Laboratory of Immune System Biology, Government of the United States of America - Laboratory of Immune System Biology, Government of the United States of America - Laboratory of Immune System Biology, Government of the United States of America - Laboratory of Immune System Biology, Government of the United States of America - Laboratory of Immune System Biology, Government of the United States of America - Laboratory of Immune System Biology, University of Pittsburgh - Department of Immunology, Government of the United States of America - National Institute of Allergy and Infectious Diseases (NIAID), Government of the United States of America - Laboratory of Immune System Biology, Government of the United States of America - Laboratory of Immune System Biology and Independent
Downloads 34 (1,263,436)

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innate immunity, adaptation, macrophages, pattern recognition receptor (PRR), toll-like receptor 4 (TLR4), Kdo2-lipid A (KLA), IL-10, temporal sensing, Inflammation