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Geneti Gaga

Government of the United States of America - National Institutes of Health (NIH)

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Selective Small Molecule Activation of Mannose Receptor CD206 Recalibrates Macrophage Function to Improve Outcomes of Cancer and Inflammatory Diseases

Number of pages: 169 Posted: 09 Sep 2025
Government of the United States of America - National Institutes of Health (NIH), Government of the United States of America - National Institutes of Health (NIH), Government of the United States of America - National Institutes of Health (NIH), Government of the United States of America - National Institutes of Health (NIH), Government of the United States of America - Thoracic Surgery Branch, Government of the United States of America - National Institutes of Health (NIH), Government of the United States of America - National Institutes of Health (NIH), Government of the United States of America - National Institutes of Health (NIH), Government of the United States of America - National Institutes of Health (NIH), Government of the United States of America - National Institutes of Health (NIH), Government of the United States of America - National Institutes of Health (NIH), Government of the United States of America - National Institutes of Health (NIH), Government of the United States of America - National Institutes of Health (NIH), Nagoya City University - Graduate School of Medical Sciences, Government of the United States of America - National Institutes of Health (NIH), Government of the United States of America - National Institutes of Health (NIH), Government of the United States of America - National Institutes of Health (NIH), Government of the United States of America - Frederick National Laboratory for Cancer Research, Government of the United States of America - National Institutes of Health (NIH), Government of the United States of America - National Institutes of Health (NIH), Government of the United States of America - National Institutes of Health (NIH), Government of the United States of America - National Institutes of Health (NIH), Government of the United States of America - National Institutes of Health (NIH), Government of the United States of America - National Institutes of Health (NIH), Government of the United States of America - National Institutes of Health (NIH), Government of the United States of America - National Institutes of Health (NIH), Government of the United States of America - National Institutes of Health (NIH), Government of the United States of America - National Institutes of Health (NIH), Government of the United States of America - Laboratory of Immune System Biology, Government of the United States of America - Frederick National Laboratory for Cancer Research, Government of the United States of America - Frederick National Laboratory for Cancer Research, Government of the United States of America - National Institutes of Health (NIH), Government of the United States of America - National Institutes of Health (NIH), Government of the United States of America - Laboratory of Immune System Biology, Government of the United States of America - National Institutes of Health (NIH), Government of the United States of America - National Institutes of Health (NIH), Government of the United States of America - National Institutes of Health (NIH), Government of the United States of America - National Institutes of Health (NIH), Government of the United States of America - National Institutes of Health (NIH), Nagoya City University - Graduate School of Medical Sciences, Government of the United States of America - National Institutes of Health (NIH), Government of the United States of America - National Institutes of Health (NIH), Government of the United States of America - National Cancer Institute and Independent
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Abstract:

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mannose receptor 1 (MRC1, CD206), CD206 small molecule agonist, tumor associated macrophages, innate immune indolence, macrophage epigenome, CD206 glycosylation, retinal fibrosis, non-alcoholic steatohepatitis (NASH)